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Drug Profile
Montelukast
* Dr. Paul Samuel
Montelukast is a selective and orally active Pharmacokinetics
leukotriene receptor antagonist that specifically Montelukast is rapidly and nearly completely inhibits the cysteinyl leukotriene CystLT1 receptor.
absorbed following oral administration. Monelukast It is available as montelukast sodium.
is more than 99% bound to plasma proteins.
Indications
Montelukast is extensively metabolized in liver andis excreted through bile. Phenobarbital, which Prophylaxis and chronic treatment of asthma in induces hepatic metabolism, decreases the AUC adults and pediatric patients of 12 months age (plasma concentration curve) of montelukast approximately 40% following a single 10mg dose;no dosage adjustment is recommended.
Allergic rhinitis ( seasonal allergic rhinitis inadults and pediatric patients 2 years of age and Pharmacodynamics
older, and perennial allergic rhinitis in adults and Montelukast causes potent inhibition of airway pediatric patients 6months of age and older cysteinyl leukotriene receptors as demonstrated by Mechanism of action
the ability to inhibit bronchoconstriction due toinhaled LTD4 in asthmatic patients. Dosage as low The cysteinyl leukotrienes (cysLTs) are potent as 5 mg causes substantial blockage of LTD4- inflammatory mediators released by mast cells and induced-bronchoconstriction. Montelukast causes basophils on activation by antigen. They are bronchodilation within 2 hours of administration; synthesized from arachadonic acid through the action these effects were additive to the bronchodilation of the enzyme 5-OH-lipoxygenase. The terminal active metabolites of this pathway are leukotriene Dosage forms
C4, leukotrieneD4, and leukotriene E4. These agents are potent inflammatory mediators and are major agents involved in the late-phase allergic response.
antagonists work through binding to receptors ontarget cells at the end organ, interfering with the ability of the cysLTs in binding and creating a physiologic effect. Several of these agents have been developed and are available in the United States for the treatment of asthma. While both montelukast and zafirlukast are FDA approved for the treatment of asthma, only montelukast is approved for the Drug Profile
efficacy to a mild non-sedating antihistamine, such as loratadine, in the reduction of nasal symptoms, although it is less effective than an intranasal corticosteroid spray. Because montelukast is age with perennial allergic rhinitis - One effective in the treatment of both upper- and lower- airway inflammation, it is an ideal medication to Contraindications
consider in the treatment of patients with concurrent allergic rhinitis and asthma. It can be used alone or in combination with other medications for bothdiseases, and can also be considered as add-on Adverse effects
therapy in patients with persistent symptoms despite Usually none. In a 12 week placebo-controlled clinical study, drug related headache and abdominal Reference
Literature review
treatment of allergic rhinitis: an evidence-based Several studies have demonstrated the efficacy of montelukast in the treatment of both seasonal and John H. Krouse. Allergic Rhinitis-Current perennial allergic rhinitis, and a recent review outlines Pharmacotherapy. Otolaryngologic Clinics of its benefits1. In general, montelukast has comparable

Source: http://imakmj.com/articles/09-Drug-Jan2011.pdf

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